Search results for "cerebral atrophy"

showing 10 items of 10 documents

Activation-Induced Rigidity in Neurologically and Cognitively Healthy Individuals Aged 18-90 Years: A Cross-Sectional Study.

2021

Background: Rigidity is a key clinical feature of Parkinson’s disease (PD), but in a very early phase of the disease it may be absent and can be enhanced through active movements of the arm contralateral to the one being tested. Objective: To evaluate in a large cohort of neurologically and cognitively healthy (NCH) subjects aged 18–90 years if activation-induced rigidity (AR) is present in all age classes, and if there are biological differences between subjects showing AR (AR+) and not showing AR (AR-). Methods: 2,228 NCH subjects categorized as young adult (18–44 years), adult (45–64 years), elderly (65–74 years), and old/oldest-old (75–90 years) were included in the analysis, and underw…

Adultmedicine.medical_specialtyParkinson's diseaseAdolescentCross-sectional studyAudiologylacunesProdromal phaseYoung AdultCellular and Molecular NeuroscienceLateral ventriclesCognitionAtrophyHumansMedicinehealthy aging subjectscaudate atrophyYoung adultAgedglobal cerebral atrophyAged 80 and overActivation-induced rigiditybusiness.industryBrainParkinson DiseaseMiddle Agedwhite matter hyperintensitiesmedicine.diseaseMagnetic Resonance ImagingHyperintensityCross-Sectional StudiesHealthy individualsSettore MED/26 - NeurologiaNeurology (clinical)Atrophybusiness
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Nociceptive Primitive Reflexes in Neurologically and Cognitively Healthy Aging Subjects

2019

ABSTRACT:Background:To assess the prevalence of three nociceptive primitive reflexes (nPR), i.e., glabellar tap, snout reflex, and palmomental reflex, in neurologically and cognitively healthy (NCH) aging subjects.Objective:To investigate whether nPR are cross-sectionally associated with white matter hyperintensities (WMH), lacunes, atrophy of the caudate nuclei, and global brain atrophy.Methods:A total of 1246 NCH subjects aged 45–91 years were included in the study and underwent standard brain MRI. Atrophy of the caudate nuclei and global brain atrophy were assessed through the bicaudate ratio (BCr) and lateral ventricles to brain ratio (LVBr), respectively. WMH were assessed through visu…

MalePrimitive reflexesmedicine.medical_specialtyCaudate atrophyLacuneNeuropsychological Testsbehavioral disciplines and activitiesPalmomental reflexHealthy Aging03 medical and health sciencesLateral ventriclesNociceptive primitive reflexesCognition0302 clinical medicineAtrophyInternal medicineSnout reflexGlobal brain atrophyActivities of Daily LivingWhite matter hyperintensitiesLacunesmedicineHumansCognitive DysfunctionCognitive declineAgedPain Measurement030304 developmental biologyHealthy aging subjectsAged 80 and over0303 health sciencesGlobal cerebral atrophybusiness.industryHealthy aging subjectBrainNociceptive primitive reflexeGeneral MedicineMiddle Agedmedicine.diseaseHyperintensityNeurologyCardiologyFemaleNeurology (clinical)business030217 neurology & neurosurgeryCanadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques
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Dyskephalie-Katarakt-Hypotrichose-Syndrom (Synonyma: DCH; Ullrich-Fremerey-Dohna; Hallermann-Streiff; Francois)

1976

In a 43-year-old man dyscephalia, cataracta congenita, and hypotrichosis were the outstanding features. These signs were first described in 1953 by Ullrich and Fremerey-Dohna as a clinical entity. Since 1958 the DCH syndrome was published under the synonyms of “Francois syndrome” and of “Hallermann-Streiff syndrome”. However, as these authors did not add any essential details relevant for the classification of the syndrome we prefer to retain the term “Ullrich-Fremerey-Dohna syndrome”. In our case in addition to the above mentioned and well known manifestations, extrapyramidal hyperkinesia of the choreoanthetotic type and servere mental deficiency accompanied by mild cerebral atrophy (revea…

Cerebral atrophyPediatricsmedicine.medical_specialtyFrancois syndromebusiness.industrymedicine.diseaseMental deficiencyNeurologymedicineHypotrichosisNeurology (clinical)medicine.symptombusinessPsychiatryHyperkinesiaJournal of Neurology
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Apoe genotypes and brain imaging classes in normal cognition, mild cognitive impairment, and alzheimer’s disease: A longitudinal study

2020

Objective: To evaluate in 419 stroke-free cognitively normal subjects (CN) aged 45-82 years covering during a long prospective study (11.54 ± 1.47 years) the preclinical to dementia spectrum: 1) the distribution of small vessel disease (V) and brain atrophy (A) aggregated as following: V−/A−, V−/A+, V+/A−, V+/A+; 2) the relationship of these imaging classes with individual apolipoprotein E (APOE) genotypes; 3) the risk of progression to Alzheimer Disease (AD) of the individual APOE genotypes. Methods: Participants underwent one baseline (t0), and 4 clinical and neuropsychological assessments (t1,t2,t3, and t4). Brain MRI was performed in all subjects at t0, t2, t3 and t4.. White matter hyp…

MaleApolipoprotein Emedicine.medical_specialtyGenotypeApolipoprotein E4NeuroimagingNeuropsychological TestsAPOE genotypes Brain imaging classes Caudate atrophy Global cerebral atrophy Lacunes White matter hyperintensities Aged Aged 80 and over Alzheimer Disease Apolipoprotein E4 Apolipoproteins E Brain Case-Control Studies Cognitive Dysfunction Disease Progression Female Genotype Humans Longitudinal Studies Magnetic Resonance Imaging Male Middle Aged Neuroimaging Neuropsychological Tests Risk Factorsbrain imaging classesApolipoproteins EAtrophyNeuroimagingAlzheimer DiseaseRisk FactorsInternal medicineGlobal brain atrophymedicineHumansDementiaCognitive DysfunctionLongitudinal Studiescaudate atrophyAgedglobal cerebral atrophyAged 80 and overAPOE genotypesbusiness.industryNeuropsychologyBrainMiddle Agedwhite matter hyperintensitiesmedicine.diseaseMagnetic Resonance ImagingHyperintensityNeurologyCase-Control StudiesDisease ProgressionCardiologyFemaleNeurology (clinical)Alzheimer's diseasebusiness
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Isolated, Subtle Neurological Abnormalities in Mild Cognitive Impairment Types.

2019

Isolated, subtle neurological abnormalities (ISNA) are commonly seen in aging and have been related to cerebral small vessel disease (SVD) and subcortical atrophy in neurologically and cognitively healthy aging subjects.To investigate the frequency of ISNA in different mild cognitive impairment (MCI) types and to evaluate for each MCI type, the cross-sectional relation between ISNA and white matter hyperintensities (WMH), lacunes, caudate atrophy, and ventricular enlargement.One thousand two hundred fifty subjects with different MCI types were included in the analysis and underwent brain magnetic resonance imaging. WMHs were assessed through two visual rating scales. Lacunes were also rated…

Apolipoprotein EPrimitive reflexesmedicine.medical_specialtyIsolated subtle neurological abnormalities Mild cognitive impairment types White matter hyperintensities Lacunes Caudate atrophy Global cerebral atrophyPopulationNeurological examinationlacunesLateral ventriclesISNAAtrophycerebral atrophyInternal medicinemental disordersmedicineDementiacaudate atrophyeducationeducation.field_of_studymedicine.diagnostic_testbusiness.industryGeneral Medicinemedicine.diseaseWMHMCIHyperintensityNeurologyCardiologySettore MED/26 - NeurologiaNeurology (clinical)businessThe Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques
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Morphologic diagnosis in neuronal ceroid lipofuscinosis.

1997

Morphologic pathology in NCL is marked by two processes, the interaction of which has not yet been completely clarified: 1) degeneration of nerve cells, foremost in the cerebral cortex, resulting in considerable cerebral atrophy in early childhood forms, likely responsible for clinical and neuroradiological findings; 2) widespread accumulation of autofluorescent lysosomal lipopigments of varying ultrastructure, the demonstration of which is still largely responsible for diagnostic recognition of an individual patient's NCL. Numerous tissues and organs are available for biopsy, among them brain (historical), rectum (still favoured by some), skeletal muscle and peripheral nerves (largely by c…

AdultPathologymedicine.medical_specialtyConjunctivaAdolescentBiopsyAutopsyAtrophyNeuronal Ceroid-LipofuscinosesBiopsyMedicineHumansTissue DistributionChildCerebral atrophyCerebral Cortexmedicine.diagnostic_testbusiness.industryInfantGeneral MedicinePigments Biologicalmedicine.diseaseLipidsmedicine.anatomical_structureCerebral cortexChild PreschoolPediatrics Perinatology and Child HealthNerve DegenerationNeuronal ceroid lipofuscinosisNeurology (clinical)Morphologic diagnosisAtrophybusinessLysosomesNeuropediatrics
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Lifespan Changes of the human brain in Alzheimer's disease

2018

[EN] Brain imaging studies have shown that slow and progressive cerebral atrophy characterized the development of Alzheimer's Disease (AD). Despite a large number of studies dedicated to AD, key questions about the lifespan evolution of AD biomarkers remain open. When does the AD model diverge from the normal aging model? What is the lifespan trajectory of imaging biomarkers for AD? How do the trajectories of biomarkers in AD differ from normal aging? To answer these questions, we proposed an innovative way by inferring brain structure model across the entire lifespan using a massive number of MRI (N = 4329). We compared the normal model based on 2944 control subjects with the pathological …

0301 basic medicineMaleAgingLongevityHippocampuslcsh:MedicineTrastorns de la cognició en la vellesaAmygdalaArticle03 medical and health sciencesLateral ventricles0302 clinical medicineNeuroimagingmedicine[INFO.INFO-IM]Computer Science [cs]/Medical ImagingHumanslcsh:ScienceAgedCerebral atrophyAged 80 and overMultidisciplinarybusiness.industryNeurodegenerationlcsh:RBrainTrastorns de la memòriaHuman brainMiddle Agedmedicine.disease3. Good health030104 developmental biologymedicine.anatomical_structureFISICA APLICADADisease Progressionlcsh:QFemaleAbnormalitybusinessNeuroscience030217 neurology & neurosurgery
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Association Between Atrophy of the Caudate Nuclei, Global Brain Atrophy, Cerebral Small Vessel Disease and Mild Parkinsonian Signs in Neurologically …

2017

Background: Mild Parkinsonian signs (MPS) are commonly seen in aging, and have been related to cerebral Small Vessel Diseases (SVD) with no univocal results. Objective: The aim of this study was to investigate the cross-sectional relation between MPS and White Matter Hyperintensities (WMH), lacunes, caudate atrophy, and global cerebral atrophy in a large cohort of Neurologically and Cognitively Healthy (NCH) aging individuals. Method: 1,219 NCH individuals were included in the analysis, and underwent standard brain MRI. The items of the motor section of the Unified Parkinson’s Disease Rating Scale were used to evaluate tremor, rigidity, bradykinesia, and gait/balance/axial dysfunction. Cau…

Male0301 basic medicinemedicine.medical_specialtyNeurologyNeuropsychological TestsCohort Studies03 medical and health sciencesLateral ventricles0302 clinical medicineAtrophyInternal medicineGlobal brain atrophyBasal gangliaHumansMedicineCognitive declineAgedUltrasonographyAged 80 and overCerebral atrophybusiness.industryHeartParkinson DiseaseMiddle AgedMental Status and Dementia Testsmedicine.diseaseMagnetic Resonance ImagingHyperintensityCross-Sectional Studies030104 developmental biologyNeurologyCerebral Small Vessel DiseasesCardiologyFemaleNeurology (clinical)AtrophyCaudate Nucleusbusiness030217 neurology & neurosurgeryCurrent Alzheimer Research
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When Does Alzheimer′s Disease Really Start? The Role of Biomarkers

2021

While Alzheimer’s disease (AD) classical diagnostic criteria rely on clinical data from a stablished symptomatic disease, newer criteria aim to identify the disease in its earlier stages. For that, they incorporated the use of AD’s specific biomarkers to reach a diagnosis, including the identification of Aβ and tau depositions, glucose hypometabolism, and cerebral atrophy. These biomarkers created a new concept of the disease, in which AD’s main pathological processes have already taken place decades before we can clinically diagnose the first symptoms. Therefore, AD is now considered a dynamic disease with a gradual progression, and dementia is its final stage. With …

0301 basic medicinemedicine.medical_specialtyad spectrumGradual progressionVariable timeInfluential PublicationsReviewDiseaseCatalysislcsh:ChemistryInorganic Chemistry03 medical and health sciences0302 clinical medicineAlzheimer DiseaseRisk FactorsmedicineHumansDementiaPhysical and Theoretical ChemistryIntensive care medicineCognitive impairmentlcsh:QH301-705.5Molecular BiologyPathologicalad dynamicimaging biomarkersSpectroscopyCerebral atrophybusiness.industryOrganic ChemistrybiomarkerscsfGeneral Medicinemedicine.diseaseComputer Science Applications030104 developmental biologylcsh:Biology (General)lcsh:QD1-999business030217 neurology & neurosurgerydementiaInternational Journal of Molecular Sciences
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Mild Parkinsonian Signs in a Hospital-based Cohort of Mild Cognitive Impairment Types: A Cross-sectional Study

2018

Background:Mild Parkinsonian Signs (MPS) have been associated with Mild Cognitive Impairment (MCI) types with conflicting results.Objective:To investigate the association of individual MPS with different MCI types using logistic ridge regression analysis, and to evaluate for each MCI type, the association of MPS with caudate atrophy, global cerebral atrophy, and the topographical location of White Matter Hyperintensities (WMH), and lacunes.Methods:A cross-sectional study was performed among 1,168 subjects with different types of MCI aged 45-97 (70,52 ± 9,41) years, who underwent brain MRI. WMH were assessed through two visual rating scales. The number and location of lacunes were also rated…

0301 basic medicineApolipoprotein EMalemedicine.medical_specialtyMild parkinsonian signsbehavioral disciplines and activitiesCohort Studies03 medical and health sciencesLateral ventricles0302 clinical medicineAtrophyParkinsonian DisordersRating scaleInternal medicinemental disordersmedicineDementiaHumansCognitive Dysfunctioncaudate atrophyProspective cohort studyAgedglobal cerebral atrophyCerebral atrophyAged 80 and overbusiness.industryMiddle Agedwhite matter hyperintensitiesmedicine.diseaseHyperintensitynervous system diseases030104 developmental biologyCross-Sectional StudiesNeurologyCardiologyFemaleNeurology (clinical)business030217 neurology & neurosurgery
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